Sunday, February 25, 2007

MGM Supplement: CoQ10 and Your Heart

In the past 30 years, the prevalence of overweight and obesity has increased sharply for both adults and children. Between 1976–1980 and 2003–2004, the prevalence of obesity among adults aged 20–74 years increased from 15.0% to 32.9%.

This increase is not limited to adults. Among young people, the prevalence of overweight increased from 5.0% to 13.9% for those aged 2–5 years, 6.5% to 18.8% for those aged 6–11 years, and 5.0% to 17.4% for those aged 12–19 years.

These increasing rates raise concern because of their implications for Americans’ health. Being overweight or obese increases the risk of many diseases and health conditions, including the following:

Hypertension
Dyslipidemia (for example, high total cholesterol or high levels of triglycerides)
Type 2 diabetes
Coronary heart disease
Stroke

Gallbladder disease
Osteoarthritis
Sleep apnea and respiratory problems
Some cancers (endometrial, breast, and colon)

Although one of the national health objectives for the year 2010 is to reduce the prevalence of obesity among adults to less than 15%, current data indicate that the situation is worsening rather than improving.

How can you prevent the things listed above from harming you? CoQ10


At least 35 controlled clinical trials in Japan, Europe and the U.S. have demonstrated the effectiveness of CoQ10 therapy in congestive heart failure, angina and ischemic heart disease, and myocardial infarction. CoQ10 levels in heart tissue decline disproportionately with age. At age 20, the heart has a higher CoQ10 level than other major organs.

At age 80 this is no longer true, with heart levels cut by more than half. CoQ10 pioneer Karl Folkers (1985), in agreement with earlier Japanese studies, found lower CoQ10 levels in patients with more severe heart disease and showed that CoQ10 supplements significantly raised blood and heart tissue levels of CoQ10 in these patients.

As the bloodstream distributes antioxidants to tissues and organs throughout the body, they do double duty protecting the blood stream's other cargo from oxidation. A case in point is LDL (low-density lipoprotein), the major cholesterol-carrying blood lipid. Until about ten years ago, excessive LDL cholesterol per se was thought to cause atherosclerosis. It is now widely accepted that LDL cholesterol must first undergo oxidation to set in motion the chain of events that ends in artery-clogging plaque. Robustantioxidant defense is therefore as important as low LDL cholesterol in maintaining arterial health.

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